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TRPV1 Activation by Capsaicin Mediates Glucose Oxidation and ATP Production Independent of Insulin

Parisa Vahidi Ferdowsi et al.  | Published on 7/27/2021
Insulin resistance (IR), a key characteristic of type 2 diabetes (T2DM), is manifested by decreased insulin-stimulated glucose transport in target tissues. Emerging research has highlighted transient receptor potential cation channel subfamily V member (TRPV1) activation by capsaicin as a potential therapeutic target for these conditions. However, there are limited data on the effects of capsaicin on cell signalling molecules involved in glucose uptake. Read more here.

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